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Scavenger receptors in good cholesterol may allow SARS-CoV-2 into human body, claims study
FirstpostHigh-density lipoprotein is a type of cholesterol that has the reputation of being good for you. However, a new study published in the journal Nature Metabolism suggests that the SARS-CoV-2 virus, which causes COVID-19 infection, uses the body’s cholesterol synthesis system to enter the host cells. The study, conducted by researchers at the Academy of Military Medical Sciences in China, shows that SARS-CoV-2 actually hijacks the essentially healthy process through which HDL works to gain entry into the body. HDL, scavenger receptors and COVID-19 The study explains that the angiotensin-converting enzyme 2 receptor binds with the SARS-CoV-2 virus through a receptor-binding domain. On the other hand, the researchers found that depletion of HDL cholesterol in the cellular membranes inhibits the SARS-CoV-2 infection, ultimately suggesting that further research into blocking SR-B1 and other susceptible HDL components could potentially provide avenues to treat and prevent COVID-19 infection.
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